Duration of Contact, Type of Leprosy, And Floor Condition As Risk Factors For Leprosy Sero-positivity
(1) Department of Public Health Nutrition Faculty of Public Health Diponegoro University Semarang, Indonesia
(2) Department of Epidemiology and Tropical Diseases Faculty of Public Health Diponegoro University Semarang, Indonesia
(3) Department of Epidemiology and Tropical Diseases Faculty of Public Health Diponegoro University Semarang, Indonesia
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Adalid-Peralta, L. et al. 2011. Mechanisms underlying the induction of regulatory T cells and its relevance in the adaptive immune response in parasitic infections. Int J Biol Sci, 7(9) : 1412–1426.
Barreto, J.G. et al. 2011. Anti-PGL-I seroepidemiology in leprosy cases: household contacts and school children from a hyperendemic municipality of the Brazilian Amazon. Leprosy review, 82(4) : 358–70.
Carvalho, A.P.M. et al. 2015. Factors associated with anti-phenolic glycolipid-I seropositivity among the household contacts of leprosy cases. BMC infectious diseases, 15 : 219.
Desikan, K. V, 1977. Viability of Mycobacterium leprae outside the human body. Leprosy review, 48(4) : 231
Desikan, K. V & Sreevatsa, 1995. Extended studies on the viability of Mycobacterium leprae outside the human body. Leprosy review, 66(4) : 287–95. Available at: http://www.ncbi.nlm.nih.gov/pubmed/8637382 [Accessed March 31, 2017].
Douglas, J.T. et al. 2004. Prospective study of serological conversion as a risk factor for development of leprosy among household contacts. Clinical and Diagnostic Laboratory Immunology, 11(5) : 897–900.
Düppre, N.C. et al. 2012. Impact of PGL-I Seropositivity on the Protective Effect of BCG Vaccination among Leprosy Contacts: a Cohort Study. PLoS Neglected Tropical Diseases, 6(6) : 1711.
Durrheim, D.N. et al. 2002. Leprosy in Mpumalanga Province, South Africa--eliminated or hidden? Leprosy Review, 73(4) : 326–33.
Feenstra, S.G. et al. 2011. Recent Food Shortage Is Associated with Leprosy Disease in Bangladesh: A Case-Control Study R. O. Phillips, ed. PLoS Neglected Tropical Diseases, 5(5) : 1029
Fitness, J., Tosh, K. & Hill, A.V.S., 2002. Genetics of susceptibility to leprosy. Genes and Immunity, 3(8) : 441–453.
Frota, C.C. et al. 2010. Seropositivity to Anti-Phenolic Glycolipid-I in Leprosy Cases, Contacts and No Known Contacts of Leprosy in an Endemic and a Non-endemic Area in Northeast Brazil. Transactions of the Royal Society of Tropical Medicine and Hygiene, 104(7) : 490–5.
Goulart, I.M.B. et al. 2008. Risk and Protective Factors for Leprosy Development Determined by Epidemiological Surveillance of Household Contacts. Clinical and Vaccine Immunology : CVI, 15(1) : 101
Hegazy, A.A. et al. 2002. Leprosy in a High-Prevalence Egyptian Village: Epidemiology and Risk Factors. International Journal of Dermatology, 41(10) : 681
Kerr-Pontes, L.R. et al. 2006. Socioeconomic, Environmental, and Behavioural Risk Factors for Leprosy in North-east Brazil: Results of a Case-Control Study. International Journal of Epidemiology, 35(4) : 994–1000.
Lastória, J.C. & Abreu, M.A.M.M. de, 2014a. Leprosy: Review of the Epidemiological, Clinical, and Etiopathogenic Aspects - Part 1. Anais Brasileiros de Dermatologia, 89(2) : 205–18.
Lastória, J.C. & Abreu, M.A.M.M. de, 2014b. Leprosy: review of the epidemiological, clinical, and etiopathogenic aspects - part 1. Anais brasileiros de dermatologia, 89(2) : 205
Lavania, M. et al. 2008. Detection of Viable Mycobacterium Leprae in Soil Samples: Insights into Possible Sources of Transmission of leprosy. Infection, Genetics and Evolution, 8(5) : 627–631.
Matsuoka, M. et al. 2017. Mycobacterium Leprae DNA in Daily Using Water as a Possible Source of Leprosy Infection. Indian Journal of Leprosy, 71(1) : 61.
Moet, F. et al. 2004. Risk Factors for Development of Clinical Leprosy among Contacts and Their Relevance for Targeted Interventions. Leprosy Review, 75(4) : 310–326.
Moet, F.J. et al. 2006. Physical Distance, Genetic Relationship, Age, and Leprosy Classification Are Independent Risk Factors for Leprosy in Contacts of Patients with Leprosy. The Journal of Infectious Diseases, 193(3) : 346–353.
Muharry, A., 2014. Faktor Risiko Kejadian Kusta. KEMAS Jurnal Kesehatan Masyarakat, 9(2) : 174–182
Nagar, N., 2007. Serum Profile Of Zinc , Copper And Magnesium In Lepromatous Leprosy – A Case Control Study.
Penna, M.L.F. et al. 2016. Anti-PGL-1 Positivity as a Risk Marker for the Development of Leprosy among Contacts of Leprosy Cases: Systematic Review and Meta-analysis. PLoS Neglected Tropical Diseases, 10(5) : 0004703.
Rahfiludin, M.Z. et al. 2007. The Difference of Anti Phenolic Glycolipid-1 (PGL-1) Immunoglobulin-M (IgM) Level and Nutritional Intake in Subclinical Leprosy Patients who Reside at Home and in The Orphanage. Medical Journal of Indonesia, 16(4) : 224
Reinar Liv, M. et al. 2008. Interventions for Skin Changes Caused by Nerve Damage in Leprosy. Cochrane Database of Systematic Reviews, (3)
Schuring, R.P. et al. 2006. Association between Anti-pGL-I IgM and Clinical and Demographic Parameters in Leprosy. Leprosy Review, 77(4) : 343
Silva, R.C. da et al. 2008. The Result Patterns of ML Flow and ELISA (PGL-I) Serologic Tests in Leprosy-Endemic and Non-Endemic Areas. Revista da Sociedade Brasileira de Medicina Tropical, 41 Suppl 2 : 19–22.
Sujagat, A. et al. 2015. Penemuan Kasus Infeksi Kusta Subklinis pada Anak melalui Deteksi Kadar Antibodi (IgM) anti PGL-1. Kesmas: National Public Health Journal, 10(2) : 74.
World Health Organization, 2012. Global leprosy situation, Geneva: World Health Organization.
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